In Gerrkek’s writing he tends to us personal experiences to support his argument   His personality tends to show in his writing as well, adding jokes to lighten the subject and as he describes himself as a “laughing at everything” and “optimistic”, the reader is able to see a hint of these qualities in his writing but does very well in supporting the argument.  His writing fallows a safe structure, supporting both sides of the argument and concluding with the side he agrees with; resulting in his papers being well planned and organized.

Hilary

Hilary seems to be shy or a little awkward and shows a little insecurity about her writing in the beginning of the semester.  But like she says in her first paper, being introduced to Elbow and Bartholmae seems to have helped develop her writing a little more and has made her later writings in the semester stronger.  The last few writings she shares shows improvement and she seems to become more open and more comfortable with her writing.

Jamie

Jamie is very open with her writing.  She tends to draw her readers with a “hook” by telling a story of personal experience or another related narrative.  She seems to completely grasp the knowledge of the topic of argumentative discourse and applies her knowledge through personal experiences and develops an argument of academic discourse through her comprehension of the general topic.

Kyle

In Kyle’s “About Page” he describes his writing as “strange” and “outside the norm” and in the beginning of the semester Kyle’s writing seems to be a bit random with no organizational structure, but this seems to work well for Kyle.  He always seems to somehow go back to the point of the paper making the whole of the paper seem rather smooth and his random acts necessary in making his point.

My new client had a different case of Alzheimer’s which make you think she didn’t even have the disease but maybe schizophrenia with dementia or another disease undiagnosed but, her doctor’s insisted on Alzheimer’s with maybe some signs of schizophrenia, that may explain her unique behavior. Alzheimer’s can be very complex but with my background in the nursing home and a few biology and pre-nursing courses, I had a good understanding of what the disease is and a few of the causes during the disease.

There are three parts to your brain the cerebrum, controls movement and is involved in remembering, problem solving, thinking and feeling. The cerebellum controls coordination and balance and the third part of your brain is the brain stem which, connects the brain to the spinal cord and controls automatic functions (breathing, digestion, heart rate and blood pressure). The cerebral cortex is a structure within the brain that plays a key role in memory, attention, perceptual awareness, thought, language, and consciousness. When these systems began to fail the mind and body loose complete control, and your life, as you know it, beings to fade. Among some of the problems associated with brain disorders is Alzheimer’s disease. A disease that leads to nerve cell death and tissue loss throughout the brain. Over time, the brain shrinks dramatically, affecting nearly all its functions.

In the adult brain there are about 100 billion nerve cells or neuron branches that connect at more than 100 trillion points. These nerve cells and connections create cell signaling which create tiny electrical charges that signal to form memories and thought movement through an individual nerve cell connecting together at synapses. When a charge reaches a synapse, it may trigger release of tiny bursts of chemicals called neurotransmitters that travel across the synapse, carrying signals to other cells. In an Alzheimer affected brain, neurons are the chief type of cell destroyed, disrupting both the way electrical charges travel within cells and the activity of neurotransmitters. The cortex shrivels up, damaging areas involved in thinking, planning, and remembering. Shrinkage is especially severe in the hippocampus, an area of the cortex which functions in formation of new memories. Ventricles, fluid-filled spaces, grow larger, and abnormal clusters of protein fragments build up between nerve cells. These dead and dying nerve cells contain tangles, which are made up of twisted strands of another protein.

Our time and our experiences create patterns of activity and explain, at the cellular level, how our brains code our thoughts, memories, skills and gives a sense of who we are. Specific activity patterns change throughout life as we meet new people, have new experiences and acquire new skills. The patterns also change when Alzheimer’s disease disrupts nerve cells and their connections to one another. Sciences are not absolutely sure what causes cell death and tissue loss in the Alzheimer brain. Doctor’s can only make a diagnosis of “possible” or “probable” Alzheimer’s Disease while a person is still alive. The only true diagnosis is through autopsy which revels the dramatic shrinkage, especially of the cortex and through a microscope doctors are able to see the widespread fatty deposits in small blood vessels, dead and dying brain cells, and abnormal deposits in and around the brain cells.

When you spend eight, twelve, and 24 hours days with an AD patient you begin to see their cognitive functions increase and decrease each day. One day someone may remember how to brush their teeth and hair and the next become extremely confused and try brushing teeth with their hair brush and hair with a toothbrush. My new client had an extremely unique case of AD. She was in the very last stages of the disease which suggest that her brain was almost completely gone. Her mood and behavior were very child-like and was extremely talented in masking this behavior. A complete change would occur when we were in public or when a visitor came over. This was a completely different experience for me, for I was used to child-like behavior and withdrawal from friends and family. In my new client’s case she changed in personality and behavior and engaged in cover-up around old friends and family. There were no signs of her showing any symptoms of the disease. She maintained her eyesight, mobility, was able to perform familiar tasks.

Most AD patients are aware of their decreasing ability of cognitive function but Jane was in complete denial and became excessively upset when told she had AD. Her doctors have suggested some schizophrenia and still insist in her diagnosis of Alzheimer’s. Jane was a fan of hovering with some tendencies to nag and pick out every little detail while I cleaned or did any kind of work around the house. This was very irritating but may have been due to her OCD behavior and often treated me and other caregivers as servants. I was often bossed around and told to complete tasks beyond ridiculous: iron sheets, re-making her bed until perfect or until she reached satisfaction, washing dishes before putting them into the dishwasher. While tending to her garden (as a favor) she would often make me show her the root of the weed to make sure I got the entire thing out of the ground, if not, she’d tell me to dig up the rest.

She had the tendency to make fun of overweight people, engaging in laughter and very inappropriate in public saying, “Would you look at that,” and “My, that is gross,” usually laughing while saying these things. Often she engaged in conversations with herself or with someone she claimed was in the room. These conversations often took place alone in her bedroom and while in bed before falling asleep and sometimes during the day. Before beginning this job I was told that people often heard strange noises during the night and saw strange shadows throughout the house and her daughter mentioned feeling her father’s presence in the house. When asked who she is speaking to she laughs and says, “Well, you know” and goes off on a completely different subject.

She had some random, “upset”, outbursts. She would grow angry about something unknown, usually seen on television. “That just makes me so angry” and hit’s the arms of her chair. Even when nothing on television is showing that would set off hostile behavior. Although Jane was sometimes difficult to deal with she was often very happy and very fun to work with. I enjoyed my time spent with her and gained all more experience with working with the elderly and with Alzheimer’s disease (or a disease unnamed).

Model:

The gallows stood in a small yard, separate from the main grounds of the prison and overgrown with tall prickly weeds. –George Orwell, Burmese Days

Imitation:

The doctor waited patiently, his stethoscope hanging around his neck, symbolizing his authoritative position.

Model:

He went through the narrow alley of Temple Bar quickly, muttering to himself that they could all go to hell because he was going to have a good night of it.  –James Joyce, “Counter Parts”

Imitation:

He stared at his patient, contemptuously, wondering if he wanted to waste his time on a man that would not make it.

Model:

To regain the stage in its own character, not as a mere emulation of prose, poetry must find its own poetic way to the mastery the stage demands—the mastery of action.  –Archibald MacLeish, “The Poet as Playwright”

Imitation:

To regain his professionalism, not only for the sake of his patient, he straightened his tie and began to recite the diagnosis.

Model:

The gallows stood in a small yard, separate from the main grounds and overgrown with tall prickly weeds.  –George Orwell, Burmese Days

Variation:

In a small yard stood the gallows, overgrown with tall prickly weeds and separate from the main grounds.

Alternate Expressions:

Separate from the main grounds stood the gallows in small yard, overgrown with tall prickly weeds.

 Overgrown with tall prickly weeds, stood the gallows in a small yard; separate from the main grounds.

Early and Late Stages of Life

            At a very young age I always knew I wanted to be a doctor or a nurse.  I remember playing “doctor” with my dolls and when preparing to take my test as a certified nursing assistant I practiced on my little sisters and took my parents blood pressures as often as they would let me.  When I received my certification I started work at a nursing home in Greeley Colorado.  It wasn’t any different from the clinical hours I completed while taking my C.N.A courses except, I was able to develop a relationship with my residents.  I worked at this nursing home for two years during which I saw a variety of different diseases, dementia, sun downers syndrome, and Alzheimer’s disease.  By the time I left the nursing home, to start a job with a private client, more than half the residents that were there when I started had passed.

            My new client had a different case of Alzheimer’s which make you think she didn’t even have the disease but maybe schizophrenia with dementia or another disease undiagnosed but, her doctor’s insisted on Alzheimer’s with maybe some signs of schizophrenia, that may explain her unique behavior.  Alzheimer’s can be very complex but with my background in the nursing home and a few biology and pre-nursing courses, I had a good understanding of what the disease is and a few of the causes during the disease.

            There are three parts to your brain the cerebrum, controls movement and is involved in remembering, problem solving, thinking and feeling.  The cerebellum controls coordination and balance and the third part of your brain is the brain stem which, connects the brain to the spinal cord and controls automatic functions (breathing, digestion, heart rate and blood pressure).  The cerebral cortex is a structure within the brain that plays a key role in memory, attention, perceptual awareness, thought,  language, and consciousness.  When these systems begin to fail, the mind and body loose complete control, and your life, as you know it, beings to fade.  Among some of the problems associated with brain disorders is Alzheimer’s disease; a disease that leads to nerve cell death and tissue loss throughout the brain.  Over time, the brain shrinks dramatically, affecting nearly all its functions.

            In the adult brain there are about 100 billion nerve cells or neuron branches that connect at more than 100 trillion points.  These nerve cells and connections create cell signaling which create tiny electrical charges that signal to form memories and thought movement through an individual nerve cell connecting together at synapses.  When a charge reaches a synapse, it may trigger release of tiny bursts of chemicals called neurotransmitters that travel across the synapse, carrying signals to other cells.  In an Alzheimer affected brain, neurons are the chief type of cell destroyed, disrupting both the way electrical charges travel within cells and the activity of neurotransmitters.  The cortex shrivels up, damaging areas involved in thinking, planning, and remembering.  Shrinkage is especially severe in the hippocampus, an area of the cortex which functions in formation of new memories.  Ventricles, fluid-filled spaces, grow larger, and abnormal clusters of protein fragments build up between nerve cells.  These dead and dying nerve cells contain tangles, which are made up of twisted strands of another protein.

            Our time and our experiences create patterns of activity and explain, at the cellular level, how our brains code our thoughts, memories, skills and gives a sense of who we are. Specific activity patterns change throughout life as we meet new people, have new experiences and acquire new skills.  The patterns also change when Alzheimer’s disease disrupts nerve cells and their connections to one another.  Sciences are not absolutely sure what causes cell death and tissue loss in the Alzheimer brain.  Doctor’s can only make a diagnosis of “possible” or “probable” Alzheimer’s Disease while a person is still alive.  The only true diagnosis is through autopsy which revels the dramatic shrinkage, especially of the cortex and through a microscope doctors are able to see the widespread fatty deposits in small blood vessels, dead and dying brain cells, and abnormal deposits in and around the brain cells.

              When you spend eight, twelve, and 24 hours days with an AD patient you begin to see their cognitive functions increase and decrease each day.  One day someone may remember how to brush their teeth and hair and the next become extremely confused and try brushing teeth with their hair brush and hair with a toothbrush.  My new client had an extremely unique case of AD.  She was in the very last stages of the disease which suggest that her brain was almost completely gone.  Her mood and behavior were very child-like and was extremely talented in masking this behavior.  A complete change would occur when we were in public or when a visitor came over.  This was a completely different experience for me, for I was used to child-like behavior and withdrawal from friends and family.  In my new client’s case she changed in personality and behavior and engaged in cover-up around old friends and family.  There were no signs of her showing any symptoms of the disease.  She maintained  her eyesight, mobility, was able to perform familiar tasks.

            Most AD patients are aware of their decreasing ability of cognitive function but Jane was in complete denial and became excessively upset when told she had AD.  Her doctors have suggested some schizophrenia and still insist in her diagnosis of Alzheimer’s.  Jane was a fan of hovering with some tendencies to nag and pick out every little detail while I cleaned or did any kind of work around the house.  This was very irritating but may have been due to her OCD behavior and often treated me and other caregivers as servants.  I was often bossed around and told to complete tasks beyond ridiculous:  iron sheets, re-making her bed until perfect or until she reached satisfaction, washing dishes before putting them into the dishwasher.  While tending to her garden (as a favor) she would often make me show her the root of the weed to make sure I got the entire thing out of the ground, if not, she’d tell me to dig up the rest.

            She had the tendency to make fun of overweight people, engaging in laughter and very inappropriate in public saying, “Would you look at that,” and “My, that is gross,” usually laughing while saying these things.  Often she engaged in conversations with herself or with someone she claimed was in the room.  These conversations often took place alone in her bedroom and while in bed before falling asleep and sometimes during the day.  Before beginning this job I was told that people often heard strange noises during the night and saw strange shadows throughout the house and her daughter mentioned feeling her father’s presence in the house.  When asked who she is speaking to she laughs and says, “Well, you know” and goes off on a completely different subject.

            She had some random, “upset”, outbursts.  She would grow angry about something unknown, usually seen on television.  “That just makes me so angry” and hit’s the arms of her chair.  Even when nothing on television was showing any discomfort that would set off hostile behavior.  Although Jane was sometimes difficult to deal with she was often very happy and very fun to work with.  I enjoyed my time spent with her and gained all more experience with working with the elderly and with Alzheimer’s disease (or a disease unnamed). 

            Jane’s family became extremely difficult to work with and depended too much for little pay from the care givers working for their mother.  One particular daughter became a threat to the staff and in some cases may a have lead to life threatening.  The staff soon ended their stay with Jane and she was moved from her home by her family and placed in a nursing home.  Although I was very upset and sad to leave her I found it a good idea to get out of a situation before it become too difficult to handle.  I enjoyed my time I had with Jane and gained a great deal of experience with Alzheimer’s disease.  

In “Arts of the Contact Zone”, Mary Pratt develops a term called contact zone and refers to it to “social spaces where cultures meet, clash, and grapple with each other, often in contest of highly asymmetrical relation of power, such as colonialism, slavery, or their aftermaths as they are lived out in many parts of the world today.” (Pratt 34). Her idea of the contact zone is intended to compare and “refer to social spaces where cultures meet, clash, and grapple with each other.” The intention of the contact zone is to contrast with ideas of communities that trigger much of the thinking about language, communication, and culture that gets done in the academy. Usually seen in literature arts, dynamics of language, writing, and representation.

Pratt identifies to phenomenon’s of the contact zone, Transculturation and autoethnography. She describes the term transcultureation to “describe processes whereby members of subordinated or marginal groups select and invent for materials transmitted by a dominant or metropolitan culture” (Pratt 36). Autoethnography is described as a text “in which people undertake to describe themselves in ways that engage with representations others have made them” (Pratt 35). She argues this phenomena by using Guaman Poma’s “unreadable” text: New Chronicle and Good Government in the twentieth century. Both these literate arts are becoming more visible, more pressing, and more decipherable in today’s writing.

Pratt then goes on to explain language in communities and in contact zones by the utopian sense of language in current thinking about speech communities through imagined communities and normative assumptions of unified and homogenous speech communities and so unified and homogenous social worlds. She concludes by addressing the teaching and learning in the contact zone through education and the university.

He points out that imitation has been a well known used implement for the performance of any form education that are more likely to give rendition to imitation exercises. He addresses the theory of imitation and romanticism quoting Fiske and Spariosu suggesting that imitation is the heart of romanticism and simply that the practice imitation is refined by romanticism. He concludes that romanticis may clearly point to another form of imitation. Muckelbauer includes Farmer and Arrington’s review of scholarship on imitation pedagogy and conclude that there is no room for imitation and a thing in the past. He argues this by playing off of Nietzsche’s “God is dead; but given the way of men, there may still be caves for thousands of years in which his shadows will be shown” (Mucklebauer 64) and suggests that imitation are shadows; “shadow that are structured by imitation itself, shadows that are imitations of imitation” (64).

He addresses the question of how one finds these shadows of imitation by giving attention to McKeon’s three primary meanings of imitation and give examples of imitation theory (philosophy) and day to day teaching of imitative practices (rhetoric). He then believes that the different uses of imitation cut through each other more than the usual method of categorization implies and explains in detail his three different modes made into three parts of the same thing: I. Repetition of the Same: Reproduction, in which the subject engages in a model and attempts to produce an exact replica. II. Repetition of Difference: Variation, this being quiet common, in which the subject engages in a model and produces a repetition but in a different context; and III. Difference and Repetition: Inspiration, where the subject is inspired by other works of scholar.

In the adult brain there are about 100 billion nerve cells or neuron branches that connect at more than 100 trillion points. These nerve cells and connections create cell signaling which create tiny electrical charges that signal to form memories and thought movement through an individual nerve cell connecting together at synapses. When a charge reaches a synapse, it may trigger release of tiny bursts of chemicals called neurotransmitters that travel across the synapse, carrying signals to other cells. In an Alzheimer affected brain, neurons are the chief type of cell destroyed, disrupting both the way electrical charges travel within cells and the activity of neurotransmitters. The cortex shrivels up, damaging areas involved in thinking, planning, and remembering. Shrinkage is especially severe in the hippocampus, an area of the cortex which functions in formation of new memories. Ventricles, fluid-filled spaces, grow larger, and abnormal clusters of protein fragments build up between nerve cells. These dead and dying nerve cells contain tangles, which are made up of twisted strands of another protein.

Our time and our experiences create patterns of activity and explain, at the cellular level, how our brains code our thoughts, memories, skills and gives a sense of who we are. Specific activity patterns change throughout life as we meet new people, have new experiences and acquire new skills. The patterns also change when Alzheimer’s disease disrupts nerve cells and their connections to one another. Sciences are not absolutely sure what causes cell death and tissue loss in the Alzheimer brain. Doctor’s can only make a diagnosis of “possible” or “probable” Alzheimer’s Disease while a person is still alive. The only true diagnosis is through autopsy which revels the dramatic shrinkage, especially of the cortex and through a microscope doctors are able to see the widespread fatty deposits in small blood vessels, dead and dying brain cells, and abnormal deposits in and around the brain cells.

When you spend eight, twelve, and 24 hours days with an AD patient you begin to see their cognitive functions increase and decrease each day. One day someone may remember how to brush their teeth and hair and the next become extremely confused and try brushing teeth with their hair brush and hair with a toothbrush. My new client had an extremely unique case of AD. She was in the very last stages of the disease which suggest that her brain was almost completely gone. Her mood and behavior were very child-like and was extremely talented in masking this behavior. A complete change would occur when we were in public or when a visitor came over. This was a completely different experience for me, for I was used to child-like behavior and withdrawal from friends and family. In my new client’s case she changed in personality and behavior and engaged in cover-up around old friends and family. There were no signs of her showing any symptoms of the disease. She maintained her eyesight, mobility, was able to perform familiar tasks.

Most AD patients are aware of their decreasing ability of cognitive function but Jane was in complete denial and became excessively upset when told she had AD. Her doctors have suggested some schizophrenia and still insist in her diagnosis of Alzheimer’s. Jane was a fan of hovering with some tendencies to nag and pick out every little detail while I cleaned or did any kind of work around the house. This was very irritating but may have been due to her OCD behavior and often treated me and other caregivers as servants. I was often bossed around and told to complete tasks beyond ridiculous: iron sheets, re-making her bed until perfect or until she reached satisfaction, washing dishes before putting them into the dishwasher. While tending to her garden (as a favor) she would often make me show her the root of the weed to make sure I got the entire thing out of the ground, if not, she’d tell me to dig up the rest.

She had the tendency to make fun of overweight people, engaging in laughter and very inappropriate in public saying, “Would you look at that,” and “My, that is gross,” usually laughing while saying these things. Often she engaged in conversations with herself or with someone she claimed was in the room. These conversations often took place alone in her bedroom and while in bed before falling asleep and sometimes during the day. Before beginning this job I was told that people often heard strange noises during the night and saw strange shadows throughout the house and her daughter mentioned feeling her father’s presence in the house. When asked who she is speaking to she laughs and says, “Well, you know” and goes off on a completely different subject.

She had some random, “upset”, outbursts. She would grow angry about something unknown, usually seen on television. “That just makes me so angry” and hit’s the arms of her chair. Even when nothing on television is showing that would set off hostile behavior.